Journal of Applied Physiology
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J Appl Physiol 62: 1944-1953, 1987;
8750-7587/87 $5.00
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Journal of Applied Physiology, Vol 62, Issue 5 1944-1953, Copyright © 1987 by American Physiological Society

ARTICLES

Supramaximal exercise after training-induced hypervolemia. I. Gas exchange and acid-base balance

H. J. Green, R. L. Hughson, J. A. Thomson and M. T. Sharratt

The effect of an exercise-induced reduction in blood O2-carrying capacity on ventilatory gas exchange and acid-base balance during supramaximal exercise was studied in six males [peak O2 consumption (VO2peak), 3.98 +/- 0.49 l/min]. Three consecutive days of supramaximal exercise resulted in a preexercise reduction of hemoglobin concentration from 15.8 to 14.0 g/dl (P less than 0.05). During exercise (120% VO2peak) performed intermittently (1 min work to 4 min rest); a small but significant (P less than 0.05) increase was found for both O2 consumption (VO2) (l X min) and heart rate (beats/min) on day 2 of the training. On day 3, VO2 (l/min) was reduced 3.2% (P less than 0.05) over day 1 values. No changes were found in CO2 output and minute ventilation during exercise between training days. Similarly, short-term training failed to significantly alter the changes in arterialized blood PCO2, pH, and [HCO-3] observed during exercise. It is concluded that hypervolemia-induced reductions in O2-carrying capacity in the order of 10-11% cause minimal impairment to gas exchange and acid-base balance during supramaximal non-steady-state exercise.


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N. J. A. Berger, I. T. Campbell, D. P. Wilkerson, and A. M. Jones
Influence of acute plasma volume expansion on VO2 kinetics, VO2peak, and performance during high-intensity cycle exercise
J Appl Physiol, September 1, 2006; 101(3): 707 - 714.
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